2.7.1.86	malfunction	deletion of the POS5 gene results in respiratory deficiency, anti-oxidative dysfunction and arginine auxotrophy of cells, and the respiratory deficiency is fatal for mitochondrial function	-, 721139	
2.7.1.86	malfunction	DELTApos5 mitochondria cannot efficiently synthesize Fe-S clusters on endogenous aconitase or imported ferredoxin	-, 722693	
2.7.1.86	malfunction	yeast cells lacking the mitochondrial NADH kinase encoded by POS5 display increased sensitivity to hydrogen peroxide, a slow-growth phenotype, reduced mitochondrial function and increased levels of mitochondrial protein oxidation and mitochondrial DNA mutations	-, 705593	
2.7.1.86	physiological function	a triple mutant lacking the activities of ATP-NAD/NADH kinase Utr1, ATP-NADH kinase Pos5 and ATP-NADH kinase Yel041wp, i.e. Yef1, is viable and does not show growth defects. Presence of Utr1 protein is critical for growth in low iron medium	661696	
2.7.1.86	physiological function	the enzyme contributes to the maintenance of respiratory chain complex activities	-, 721139	
2.7.1.86	physiological function	the enzyme is required for efficient iron-sulfur cluster biogenesis in wild type mitochondria	-, 722693	
2.7.1.86	physiological function	the enzyme might have a role in waterlogging tolerance in legumes	759974