1.6.3.1	additional information	direct interaction of TLR4 with NAD(P)H oxidase 4 isozyme is essential for lipopolysaccharide-induced production of reactive oxygen species and activation of NF-kappaB	
1.6.3.1	additional information	the production of reactive oxygen species is initiated by the phygocyte NADPH oxidase	
1.6.3.1	additional information	enzyme isoform Nox3 plays an integral role in insulin-induced p42/44 MAPK signal transmission and VEGF-A production	
1.6.3.1	additional information	enzyme provides hydrogen peroxide for iodine metabolism in the thyroid	
1.6.3.1	additional information	enzyme-derived reactive oxigen species modulate cerebral vascular tone under physiological conditions	
1.6.3.1	additional information	essential role of enzyme-generated reactive oxigen species in insulin-stimulated activation of hypoxia-inducible factor 1	
1.6.3.1	additional information	formation of a complex consisting of enzyme, guanine exchange factor for Rac, betaPix, and enzyme organizer NoxO1 is a critical step in EGF-induced generation of reactive oxygen species	
1.6.3.1	additional information	plasma membrane NADPH oxidase participates in the Ni-induced production of reactive oxygen species, and Ca2+ may be involved in the oxidative stress induced by nickel in wheat roots	
1.6.3.1	additional information	reactive oxygen species generated by the transverse tubule NADPH oxidase enzyme activate via redox modification the neighboring RyR1 Ca2+ release channels	
1.6.3.1	additional information	active NAD(P)H oxidase is required for vascular endothelial growth factor activation of phosphoinositide 3-kinase-Akt-forkhead, and p38 mitogen-activated kinase, but not extracellular signal-related kinase 1/2 or c-Jnu N-terminal kinase. The permissive role of NADPH oxidase on phosphoinositide 3-kinase-Akt-forkhead signaling is mediated at post-vascular endothelial growth factor receptor levels and involves the nonreceptor tyrosine kinase Src