2.7.11.18	ATP + light chain myosin II	-	
2.7.11.18	ATP + light chain myosin II	cardiac-MLCK is essential for normal cardiac development and function in zebrafish embryos, deletion of the gene encoding cardiac-MLCK is lethal for embryos	
2.7.11.18	ATP + light chain myosin II	causes endothelial contraction	
2.7.11.18	ATP + light chain myosin II	cytokine-induced epithelial barrier dysfunction can be mediated by increased MLCK expression and subsequent myosin II regulatory light chain phosphorylation, the enzyme is involved in the inflammatory bowel disease	
2.7.11.18	ATP + light chain myosin II	MLC phosphorylation increases muscle contraction and plays a prominent role in skeletal muscle force potentiation of fast-twitch type IIb but not type I or IIa fibers, overview	
2.7.11.18	ATP + light chain myosin II	MLCK plays a key role in the control of MLC-phosphorylation status, and it modulates barrier function through its regulation of intracellular contractile machinery, overview, endothelial mechanism of MLC-dependent barrier injury in burns, overview	
2.7.11.18	ATP + light chain myosin II	myosin II activation is essential for stress fiber and focal adhesion formation, and is implicated in integrin-mediated signaling events	
2.7.11.18	ATP + light chain myosin II	regulatory myosin chain, in thick filaments, isozyme L-MLCK plays a role in cytoskeleton organization	
2.7.11.18	ATP + light chain myosin II	roles of MLCK and ROCK on myosin II activation, overview, a global, MLCK-dependent increase in myosin II cortical contractility accompanies the metaphase-anaphase transition in sea urchin eggs, overview	
2.7.11.18	ATP + light chain myosin II	the enzyme is important in regulation of shape, adhesion and migration of fibrosarcoma cells, overview