1.8.3.1 1-Ethyl-3-(3-dimethylaminopropyl)carbodiimide hydrochloride EDC 42906 1.8.3.1 2,6-dichloroindophenol inhibition of O2 consumption 670 1.8.3.1 Ag+ - 75 1.8.3.1 arsenate 100 mM, EPR spectra 255 1.8.3.1 arsenite - 397 1.8.3.1 arsenite 7.4 mM, 50% inhibition 397 1.8.3.1 Ca2+ - 15 1.8.3.1 CN- - 159 1.8.3.1 CN- more than 10 mM 159 1.8.3.1 CN- profound at low O2 concentration, not at high O2 concentration 159 1.8.3.1 CN- mechanism of inactivation 159 1.8.3.1 cytochrome c inhibition of O2 consumption 84 1.8.3.1 diethylpyrocarbonate modifies ten His per enzyme molecule 2270 1.8.3.1 EDTA 20 mM, 50% inhibition 21 1.8.3.1 ferricyanide irreversible inactivation of molybdenum center 132 1.8.3.1 ferricyanide inhibition of O2 consumption 132 1.8.3.1 heavy metal ions - 3689 1.8.3.1 Hg2+ - 33 1.8.3.1 imidazole 0.1 mM, complete inhibition 470 1.8.3.1 K2HPO4 at 26 mM 50% inhibition if cytochrome c or ferricyanide is electron acceptor 1620 1.8.3.1 K2SO4 at 22 mM 50% inhibition if cytochrome c or ferricyanide is electron acceptor 2560 1.8.3.1 KCl at 95 mM 50% inhibition if cytochrome c or ferricyanide is electron acceptor 79 1.8.3.1 KCl 30 mM, 50% inhibition 79 1.8.3.1 KF at 72 mM 50% inhibition if cytochrome c or ferricyanide is electron acceptor 2410 1.8.3.1 KNCS at 57 mM 50% inhibition if cytochrome c or ferricyanide is electron acceptor 109078 1.8.3.1 KNO3 at 78 mM 50% inhibition if cytochrome c or ferricyanide is electron acceptor 2273 1.8.3.1 KNO3 3 mM, 50% inhibition 2273 1.8.3.1 mannitol only with O2 as electron acceptor 1791 1.8.3.1 methylene blue 70% inhibition at 0.4 mM 512 1.8.3.1 additional information not inhibitory: CN- 2 1.8.3.1 additional information not inhibitory: NaN3 at 0.5 mM, NaCN at 0.5 mM 2 1.8.3.1 additional information inhibition at 400 mM salt concentrations; no inhibition by periodate or 80 mM sulfate 2 1.8.3.1 additional information maintaining animals on high tungsten, low molybdenum diet, effectively induces SOX deficiency 2 1.8.3.1 additional information maintaining the animal on low-molybdenum, high-tungsten diet, leads to an effective production of SOX deficiency 2 1.8.3.1 additional information administration of high-tungsten/low molybdenum regimen leads to deficiency in SOX 2 1.8.3.1 additional information is inactivated in the dark via a process in which a Ser residue (Ser543) in the hinge region connecting the Mo-PPT dimerization domain with the heme b5 domain is phosphorylated, followed by binding of the NIA inhibitor protein 2 1.8.3.1 N-bromosuccinimide 94% inhibition at 0.1 mM 208 1.8.3.1 N-cyclohexyl-N'-[2-(N-methylmorpholino)-ethyl]carbodiimide p-toluene sulfonate CMC 96871 1.8.3.1 N-ethyl-5-phenylisoxazolium-3'-sulfonate Woodward's reagent K 6219 1.8.3.1 NaCl at 100 mM 50% inhibition if cytochrome c or ferricyanide is electron acceptor 42 1.8.3.1 NaCl 50% inhibition at 70 mM, in Tris/HCl 20 mM, pH 8.5 42 1.8.3.1 NaCl 150 mM, 50% inhibition 42 1.8.3.1 Ni2+ stronger inhibition at pH 7.0 than at pH 3.0 38 1.8.3.1 NiCl2 0.1 mM, complete inhibition 741 1.8.3.1 nitrite nitrite inhibits sulfite-dependent cytochrome c reduction at sulfite concentrations ranging from 0.01 to 0.1 mM 168 1.8.3.1 p-chloromercuribenzoate - 43 1.8.3.1 phosphate 100 mM, EPR spectra 16 1.8.3.1 potassium nitrate 50% inhibition at 1 mM, in Tris/HCl 20 mM, pH 8.5 32713 1.8.3.1 potassium phosphate 50% inhibition at 30 mM, pH 8.5 1510 1.8.3.1 potassium phosphate 20 mM, pH 8.0, 50% inhibition 1510 1.8.3.1 RNAi abrogates sulfite oxidase expression, whereby accumulating relatively less sulfate after SO2 application and showing enhanced induction of senescence and wounding associated transcripts, leaf necrosis and chlorophyll bleaching 1580 1.8.3.1 Sodium arsenate 7 mM, 50% inhibition 3229 1.8.3.1 Sodium sulfate 20 mM, 50% inhibition 5217 1.8.3.1 sodium sulfite a high initial concentration of sodium sulfite decreases dramatically the enzyme expression 8420 1.8.3.1 sodium tungstate at pH 7.5 sodium tungstate inhibits enzyme activity as follows: 1 mM 8% inhibition, 3 mM 36% inhibition, 10 mM 49% inhibition, 50 mM complete inhibition, stronger inhibition at pH 7.0 than at pH 3.0 23741 1.8.3.1 sulfate - 160 1.8.3.1 sulfite competitive inhibitor of nitric oxide production above 0.037 mM 92 1.8.3.1 Tris-acetate 80 mM, pH 8.0, 50% inhibition 60566 1.8.3.1 Tris-HCl 100 mM, pH 8.0, 50% inhibition 2227 1.8.3.1 Tris/HCl 50% inhibition at 90 mM, pH 8.5 12717 1.8.3.1 tungstate treatment with sodium tungstate, leading to a catalytically inactive analogue by replacing molybdenum with tungsten in molybdenum cofactor because of its higher affinity constant. Determination of tolerance and impact of different concentrations of tungstate on the viability of HepG2 cells using a luciferase-based cytotoxicity assay, sodium tungstate is nontoxic up to 1000 ppm 1989 1.8.3.1 tungstate complete inhibition at 1 mM 1989