1.4.4.2 5,5'-dithiobis-(2-nitrobenzoic acid) complete inhibition 2053 1.4.4.2 aminoacetonitrile the inhibitor of GDC is able to mimic mitochondrial depolarization, hydrogen peroxide production, and cell death in response to stress or harpin treatment of cultured Arabidopsis cells 9582 1.4.4.2 Aminooxyacetate - 1374 1.4.4.2 CO2 product inhibition 28 1.4.4.2 Co2+ inhibition of glycine-CO2 exchange by binding of metal with H-protein-bound intermediate of glycine decarboxylation 23 1.4.4.2 Cu2+ inhibition of glycine-CO2 exchange by binding of metal with H-protein-bound intermediate of glycine decarboxylation 19 1.4.4.2 Fe2+ slight inhibition of glycine-CO2 exchange by binding of metal with H-protein-bound intermediate of glycine decarboxylation 25 1.4.4.2 Glycine methyl ester - 12546 1.4.4.2 harpin the bacterial elicitor, a strong inducer of reactive oxygen species and NO, inhibits GDC activity 163226 1.4.4.2 K2HPO4 inhibition of glycine-CO2 exchange reaction 1620 1.4.4.2 KCl inhibition of glycine-CO2 exchange reaction 79 1.4.4.2 Modified H-protein lipoic acid prosthetic group and cysteinyl residues modified with N-ethylmaleimide 99447 1.4.4.2 additional information inhibitors of the multienzyme complex 2 1.4.4.2 additional information inactivation after incubation with glycine in presence of aminomethyl carrier protein (H-protein), it is a suicide reaction of the P-protein as a side reaction of the glycine decarboxylation 2 1.4.4.2 additional information low CO2 concentrations decrease expression of GDC 2 1.4.4.2 N-ethylmaleimide - 49 1.4.4.2 N-ethylmaleimide complete inhibition 49 1.4.4.2 N4-methylglutamine inhibition of glycine-CO2 exchange reaction 109296 1.4.4.2 Ni2+ inhibition of glycine-CO2 exchange by binding of metal with H-protein-bound intermediate of glycine decarboxylation 38 1.4.4.2 S-nitrosoglutathione i.e. GSNO, the glycine decarboxylase complex, GDC activity is inhibited by S-nitrosoglutathione due to S-nitrosylation/S-glutathionylation of several cysteine residues, overview. The inhibition of GDC activity after GSNO treatment can be an indirect effect of ROS induced by inhibition of complex I 980 1.4.4.2 serine competitive inhibitor 930 1.4.4.2 Zn2+ inhibition of glycine-CO2 exchange by binding of metal with H-protein-bound intermediate of glycine decarboxylation 14