4.1.1.9	5-aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside	AMP-activated protein kinase activator, 2fold activation of MCD in extensor digitorum longus, reversed by protein phosphatase A	652024	
4.1.1.9	Alkaline phosphatase	treatment with alkaline phosphatase enhances MCD activity suggesting phosphorylation-related control of enzyme	649690	
4.1.1.9	AMP-activated potein kinase	AMP-activated potein kinase activates MCD by phosphorylation	691964	
4.1.1.9	DL-carnitine	stimulates	4845	
4.1.1.9	fatty acid	increases MCD expression in heart and skeletal muscle	649300	
4.1.1.9	fenofibrate	increases hepatic mRNA expression and MCD activity	649775	
4.1.1.9	iodoacetamide	malonyl-decarboxylase activity is induced by treatment of fatty acid synthetase with iodoacetamide	4848	
4.1.1.9	iodoacetamide	treatment of fatty acid synthetase with iodoacetamide induces malonyl-CoA decarboxylase activity and impairs the synthetase activity	4855	
4.1.1.9	additional information	gastrocnemius contraction activates MCD, reversed by protein phosphatase A or by omitting phosphatase inhibitors from assay mixture, increase of MCD activity after skeletal muscle contraction is likely due to its phosphorylation by AMP-activated protein kinase	652024	
4.1.1.9	additional information	high-fat feeding, fasting and streptozotocin-induced diabetes increase the plasma concentration of nonesterified fatty acids, with a concomitant increase in both heart and skeletal muscle MCD mRNA	649300