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Literature summary extracted from

  • Gangaiah, D.; Kassem, I.I.; Liu, Z.; Rajashekara, G.
    Importance of polyphosphate kinase 1 for Campylobacter jejuni viable-but-nonculturable cell formation, natural transformation, and antimicrobial resistance (2009), Appl. Environ. Microbiol., 75, 7838-7849.
    View publication on PubMedView publication on EuropePMC

Organism

EC Number Organism UniProt Comment Textmining
2.7.4.1 Campylobacter jejuni
-
-
-
2.7.4.1 Campylobacter jejuni 81-176
-
-
-

Substrates and Products (Substrate)

EC Number Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
2.7.4.1 ATP + (phosphate)n
-
 Campylobacter jejuni ADP + (phosphate)n+1
-
 ?
2.7.4.1 ATP + (phosphate)n
-
 Campylobacter jejuni 81-176 ADP + (phosphate)n+1
-
 ?

Synonyms

EC Number Synonyms Comment Organism
2.7.4.1 poly-P kinase 1
-
 Campylobacter jejuni
2.7.4.1 PPK1
-
 Campylobacter jejuni

Cofactor

EC Number Cofactor Comment Organism Structure
2.7.4.1 ATP
-
 Campylobacter jejuni

General Information

EC Number General Information Comment Organism
2.7.4.1 malfunction ppk1-knockout mutant is deficient in poly-P accumulation, which is associated with a decreased ability to form viable-but-nonculturable cells under acid stress. The ppk1-deficient mutant shows a significant increase in susceptibility to erythromycin, cefotaxime, ciprofloxacin, rifampin, polymyxin B, tetracycline, cholic acid, taurocholic acid, deoxycholic acid, ethidium bromide, and SDS Campylobacter jejuni