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Literature summary extracted from

  • Martin, P.T.; Xu, R.; Rodino-Klapac, L.R.; Oglesbay, E.; Camboni, M.; Montgomery, C.L.; Shontz, K.; Chicoine, L.G.; Clark, K.R.; Sahenk, Z.; Mendell, J.R.; Janssen, P.M.
    Overexpression of Galgt2 in skeletal muscle prevents injury resulting from eccentric contractions in both mdx and wild-type mice (2009), Am. J. Physiol. Cell Physiol., 296, C476-C488.
    View publication on PubMedView publication on EuropePMC

Application

EC Number Application Comment Organism
2.4.1.165 medicine Galgt2 overexpression inhibits muscular dystrophy in old dystrophic muscles and may have therapeutic impact in many types of muscle injury Mus musculus

Organism

EC Number Organism UniProt Comment Textmining
2.4.1.165 Mus musculus
-
-
-

Source Tissue

EC Number Source Tissue Comment Organism Textmining
2.4.1.165 skeletal muscle
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Mus musculus
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Substrates and Products (Substrate)

EC Number Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
2.4.1.165 UDP-N-acetyl-D-galactosamine + alpha-N-acetylneuraminyl-(2->3)-beta-D-galactosyl-(1->4)-beta-D-glucosyl-(1<->1)-ceramide
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Mus musculus UDP + N-acetyl-beta-D-galactosaminyl-(1->4)-[alpha-N-acetylneuraminyl-(2->3)]-beta-D-galactosyl-(1->4)-beta-D-glucosyl-(1<->1)-ceramide
-
?

Synonyms

EC Number Synonyms Comment Organism
2.4.1.165 CT GalNAc transferase
-
Mus musculus
2.4.1.165 cytotoxic T cell GalNAc transferase
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Mus musculus
2.4.1.165 Galgt2
-
Mus musculus
2.4.1.165 UDP-GalNAc:beta1,4-N-acetylgalactosaminyltransferase
-
Mus musculus