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Literature summary for 5.3.99.2 extracted from

  • Nakayama, H.; Echizen, H.; Gomi, T.; Shibuya, Y.; Nakamura, Y.; Nakano, K.; Arashi, H.; Itai, T.; Ohnishi, S.; Tanaka, M.; Orii, T.
    Urinary lipocalin-type prostaglandin D synthase: a potential marker for early gentamicin-induced renal damage? (2009), Ther. Drug Monit., 31, 126-130.
    View publication on PubMed

Application

Application Comment Organism
medicine in patients receiving long-term intravenous administration of gentamicin for the treatment of infective endocarditis, systemic clearance of gentamicin is reduced by 10% from the early to late treatment phase, wherease urinary lipocalin-type prostaglandin synthase excretion shows a significant increase. No significant changes are observed for urinary beta2-microglobulin and N-acetyl-beta-D-glucosaminidase concentrations Homo sapiens

Organism

Organism UniProt Comment Textmining
Homo sapiens
-
isoform liocalin-type prostaglandin D-synthase
-