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Literature summary for 4.4.1.1 extracted from

  • Wu, L.; Yang, W.; Jia, X.; Yang, G.; Duridanova, D.; Cao, K.; Wang, R.
    Pancreatic islet overproduction of H2S and suppressed insulin release in Zucker diabetic rats (2009), Lab. Invest., 89, 59-67.
    View publication on PubMed

Application

Application Comment Organism
additional information insulin release is impaired in diabetic animals and inhibition of abnormally increased endogenous pancreatic H2S production in diabetes may represent a novel avenue for diabetes treatment Rattus norvegicus

Inhibitors

Inhibitors Comment Organism Structure
DL-propargylglycine
-
Rattus norvegicus

Organism

Organism UniProt Comment Textmining
Rattus norvegicus
-
Zucker diabetic fatty rats, comparison with Zucker fatty and Zucker lean rats
-

Source Tissue

Source Tissue Comment Organism Textmining
beta cell
-
Rattus norvegicus
-
pancreas
-
Rattus norvegicus
-

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
L-cysteine + H2O
-
Rattus norvegicus sulfide + NH3 + pyruvate
-
?

Synonyms

Synonyms Comment Organism
CSE
-
Rattus norvegicus
cystathionine gamma-lyase
-
Rattus norvegicus

Cofactor

Cofactor Comment Organism Structure
pyridoxal 5'-phosphate
-
Rattus norvegicus