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Literature summary for 4.1.1.84 extracted from

  • Li, L.; Jing, L.; Wang, J.; Xu, W.; Gong, X.; Zhao, Y.; Ma, Y.; Yao, X.; Sun, X.
    Autophagic flux is essential for the downregulation of D-dopachrome tautomerase by atractylenolide I to ameliorate intestinal adenoma formation (2018), J. Cell Commun. Signal., 12, 689-698.
    View publication on PubMed
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Application

Application Comment Organism
medicine atractylenolide 1 reduces intestinal adenoma formation through activating autophagy machinery to downregulate D-dopachrome tautomerase Homo sapiens
medicine atractylenolide 1 reduces intestinal adenoma formation through activating autophagy machinery to downregulate D-dopachrome tautomerase. Atractylenolide 1 stimulates autophagic flux through downregulating acetylation of p53 Mus musculus

Organism

Organism UniProt Comment Textmining
Homo sapiens P30046
-
-
Mus musculus O35215
-
-

Synonyms

Synonyms Comment Organism
D-dopachrome tautomerase
-
 Homo sapiens
D-dopachrome tautomerase
-
 Mus musculus
DDT
-
 Homo sapiens
DDT
-
 Mus musculus

Expression

Organism Comment Expression
Homo sapiens atractylenolide 1 stimulates autophagic flux to downregulate expression of D-dopachrome tautomerase. Deacetylation of p53 is pivotal for the induction of autophagic flux down