Any feedback?
Please rate this page
(literature.php)
(0/150)

BRENDA support

Literature summary for 3.5.4.37 extracted from

  • Wang, Q.; Miyakoda, M.; Yang, W.; Khillan, J.; Stachura, D.L.; Weiss, M.J.; Nishikura, K.
    Stress-induced apoptosis associated with null mutation of ADAR1 RNA editing deaminase gene (2003), J. Biol. Chem., 279, 4952-4961.
    View publication on PubMed

Organism

Organism UniProt Comment Textmining
Mus musculus Q99MU3
-
-

General Information

General Information Comment Organism
malfunction ADAR1-/- homozygous embryos die at E11.0 to E12.5. Widespread apoptosis is detected in many tissues of ADAR1-/- embryos collected live at E10.5 to E11.5, despite their normal gross appearance Mus musculus
physiological function ADAR1 is required for cell survival and embryo development by protecting against stress-induced apoptosis Mus musculus