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Literature summary for 3.5.3.1 extracted from

  • Hayashi, T.; Esaki, T.; Sumi, D.; Mukherjee, T.; Iguchi, A.; Chaudhuri, G.
    Modulating role of estradiol on arginase II expression in hyperlipidemic rabbits as an atheroprotective mechanism (2006), Proc. Natl. Acad. Sci. USA, 103, 10485-10490.
    View publication on PubMedView publication on EuropePMC
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Application

Application Comment Organism
medicine oopherectomized animals treated with 0.5% cholesterol-enriched diet. Diet results in increase in plasma lipids, atheromatous lesions as well as expression of enzyme isoforms arginase I and II and an increase in cellular proliferation. Diet plus supplementation of 17beta-estradiol results in a decrease of atheromatous lesions and reduced expression of both enzyme isoforms and inducible NO synthase. Inhibiton of enzyme expression by 17beta-estradiol as mechanism in attenuating atherogenensis Oryctolagus cuniculus
nutrition oopherectomized animals treated with 0.5% cholesterol-enriched diet. Diet results in increase in plasma lipids, atheromatous lesions as well as expression of enzyme isoforms arginase I and II and an increase in cellular proliferation. Diet plus supplementation of 17beta-estradiol results in a decrease of atheromatous lesions and reduced expression of both enzyme isoforms and inducible NO synthase Oryctolagus cuniculus

Natural Substrates/ Products (Substrates)

Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
additional information Oryctolagus cuniculus enzyme may be involved in cellular proliferation in atherosclerosis, inhibiton of enzyme expression by 17beta-estradiol as mechanism in attenuating atherogenensis ?
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 ?

Organism

Organism UniProt Comment Textmining
Oryctolagus cuniculus
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 oopherectomized animals treated with 0.5% cholesterol-enriched diet. Diet results in increase in plasma lipids, atheromatous lesions as well as expression of enzyme isoforms arginase I and II and an increase in cellular proliferation. Diet plus supplementation of 17beta-estradiol results in a decrease of atheromatous lesions and reduced expression of both enzyme isoforms and inducible NO synthase
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Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
additional information enzyme may be involved in cellular proliferation in atherosclerosis, inhibiton of enzyme expression by 17beta-estradiol as mechanism in attenuating atherogenensis Oryctolagus cuniculus ?
-
 ?