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Literature summary for 3.4.17.10 extracted from
- Carboxypeptidase E/NFalpha1: A New Neurotrophic Factor against Oxidative Stress-Induced Apoptotic Cell Death Mediated by ERK and PI3-K/AKT Pathways (2013), PLoS ONE, 8, e71578.
Organism
| Organism | UniProt | Comment | Textmining |
|---|---|---|---|
| Mus musculus | - |
- |
- |
Source Tissue
| Source Tissue | Comment | Organism | Textmining |
|---|
Synonyms
| Synonyms | Comment | Organism |
|---|---|---|
| carboxypeptidase E | - |
Mus musculus |
| CPE | - |
Mus musculus |
General Information
| General Information | Comment | Organism |
|---|---|---|
| physiological function | CPE acts extracellularly through activation of ERK and AKT signaling pathways to up-regulate expression of the anti-apoptotic protein BCL-2 to mediate neuroprotection of neurons during stress | Mus musculus |
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Release 2023.1 (January 2023)
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