Literature summary for 3.4.13.22 extracted from

Lee, S.J.; Jang, J.H.; Yoon, G.Y.; Kang, D.R.; Park, H.J.; Shin, S.J.; Han, H.D.; Kang, T.H.; Park, W.S.; Yoon, Y.K.; Soh, B.Y.; Jung, I.D.; Park, Y.M.
Mycobacterium abscessus D-alanyl-D-alanine dipeptidase induces the maturation of dendritic cells and promotes Th1-biased immunity (2016), BMB Rep., 49, 554-559 .

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Application

Application	Comment	Organism
medicine	Mycobacterium abscessus is an pulmonary pathogen in humans. The enzyme induces dendritic maturation via toll-like receptor 4 and its downstream signaling pathways, such as the mitogen-activated protein kinase and nuclear factor kappa B pathways. Enzyme-treated dendritic cells stimulate the proliferation of T cells and promote Th1 polarization	Mycobacteroides abscessus

Localization

Localization	Comment	Organism	GeneOntology No.	Textmining

Organism

Organism	UniProt	Comment	Textmining
Mycobacteroides abscessus	B1MNM5	-	-
Mycobacteroides abscessus DSM 44196	B1MNM5	-	-

Source Tissue

Source Tissue	Comment	Organism	Textmining

Synonyms

Synonyms	Comment	Organism
MAB1843	-	Mycobacteroides abscessus

General Information

General Information	Comment	Organism
physiological function	Mycobacterium abscessus is an pulmonary pathogen in humans. The enzyme induces dendritic maturation via toll-like receptor 4 and its downstream signaling pathways, such as the mitogen-activated protein kinase and nuclear factor kappa B pathways. Enzyme-treated dendritic cells stimulate the proliferation of T cells and promote Th1 polarization	Mycobacteroides abscessus

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Release 2023.1 (January 2023)