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Literature summary for 3.1.1.79 extracted from

  • Ueno, M.; Suzuki, J.; Zenimaru, Y.; Takahashi, S.; Koizumi, T.; Noriki, S.; Yamaguchi, O.; Otsu, K.; Shen, W.J.; Kraemer, F.B.; Miyamori, I.
    Cardiac overexpression of hormone-sensitive lipase inhibits myocardial steatosis and fibrosis in streptozotocin diabetic mice (2008), Am. J. Physiol. Endocrinol. Metab., 294, E1109-E1118.
    View publication on PubMed

Application

Application Comment Organism
medicine cardiac HSL can be a therapeutic target for regulating diabetic cardiomyopathy Mus musculus

Cloned(Commentary)

Cloned (Comment) Organism
heart-specific HSL-overexpressing mice, transgenic (Tg) mice with heart-specific HSL overexpression are generated, and cardiac histology, function, lipid profile, and gene expressions are analyzed after induction of diabetes by streptozotocin. HSL overexpression inhibits cardiac steatosis in streptozotocin (STZ)-induced diabetic mice Mus musculus

Organism

Organism UniProt Comment Textmining
Mus musculus
-
C57BL/6 mice
-
Mus musculus C57BL/6
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C57BL/6 mice
-

Source Tissue

Source Tissue Comment Organism Textmining
adipose tissue
-
Mus musculus
-
heart
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Mus musculus
-

Synonyms

Synonyms Comment Organism
cardiac HSL
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Mus musculus
hormone-sensitive lipase
-
Mus musculus
HSL
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Mus musculus