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Literature summary for 2.7.8.45 extracted from
- Lesions in teichoic acid biosynthesis in Staphylococcus aureus lead to a lethal gain of function in the otherwise dispensable pathway (2006), J. Bacteriol., 188, 4183-4189.
Organism
| Organism | UniProt | Comment | Textmining |
|---|---|---|---|
| Staphylococcus aureus | Q2G1C1 | - |
- |
| Staphylococcus aureus NCTC 8325 | Q2G1C1 | - |
- |
Synonyms
| Synonyms | Comment | Organism |
|---|---|---|
| tarF | - |
Staphylococcus aureus |
General Information
| General Information | Comment | Organism |
|---|---|---|
| physiological function | gene product TarF is essential for viability. Staphylococcus aureus shows a lethal gain-of-function mechanism where lesions beyond the initial step in wall teichoic acid biosynthesis render Staphylococcus aureus nonviable | Staphylococcus aureus |
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Release 2023.1 (January 2023)
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