Literature summary for 2.7.7.14 extracted from

Bleijerveld, O.B.; Klein, W.; Vaandrager, A.B.; Helms, J.B.; Houweling, M.
Control of the CDPethanolamine pathway in mammalian cells: effect of CTP:phosphoethanolamine cytidylyltransferase overexpression and the amount of intracellular diacylglycerol (2004), Biochem. J., 379, 711-719.

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Cloned(Commentary)

Cloned (Comment)	Organism
cDNA encoding CTP:phosphoethanolamine cytidylyltransferase is transiently or stably transfected in CHO cells and a rat liver-derived cell line (McA-RH7777), resulting in a maximum of 26fold and 4fold increase in specific activity of CTP:phosphoethanolamine cytidylyltransferase respectively	Rattus norvegicus

Natural Substrates/ Products (Substrates)

Natural Substrates	Organism	Comment (Nat. Sub.)	Natural Products	Comment (Nat. Pro.)	Rev.	Reac.
CTP + ethanolamine phosphate	Rattus norvegicus	at physiological ethanolamine concentrations, the supply of CDPethanolamine, via the expression level of CTP:phosphoethanolamine cytidylyltransferase, together with the amount of cellular diacylglycerol available to CTP:phosphoethanolamine cytidylyltransferase are major factors regulating the flux through the CDPethanolamine pathway	diphosphate + CDP-ethanolamine	-	?

Organism

Organism	UniProt	Comment	Textmining
Rattus norvegicus	-	-	-

Source Tissue

Source Tissue	Comment	Organism	Textmining
liver	-	Rattus norvegicus	-

Substrates and Products (Substrate)

Substrates	Comment Substrates	Organism	Products	Comment (Products)	Rev.	Reac.
CTP + ethanolamine phosphate	at physiological ethanolamine concentrations, the supply of CDPethanolamine, via the expression level of CTP:phosphoethanolamine cytidylyltransferase, together with the amount of cellular diacylglycerol available to CTP:phosphoethanolamine cytidylyltransferase are major factors regulating the flux through the CDPethanolamine pathway	Rattus norvegicus	diphosphate + CDP-ethanolamine	-	?

Synonyms

Synonyms	Comment	Organism
CTP:phosphoethanolamine cytidylyltransferase	-	Rattus norvegicus

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Release 2023.1 (January 2023)