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Literature summary for 2.6.1.11 extracted from
- Mutation of the Erwinia amylovora argD gene causes arginine auxotrophy, nonpathogenicity in apples, and reduced virulence in pears (2014), Appl. Environ. Microbiol., 80, 6739-6749.
Application
| Application | Comment | Organism |
|---|---|---|
| biotechnology | the pCR2.1-argD complementation plasmid is stably maintained in the argD(1000)::Tn5 transposon mutant growing in host tissues without any antibiotic selection. The pCR2.1-argD complementation plasmid can be useful for the expression of genes, markers, and reporters in Erwinia amylovora growing in planta, without concern about losing the plasmid over time | Erwinia amylovora |
Organism
| Organism | UniProt | Comment | Textmining |
|---|---|---|---|
| Erwinia amylovora | - |
- |
- |
| Erwinia amylovora HKN06P1 | - |
- |
- |
Synonyms
| Synonyms | Comment | Organism |
|---|---|---|
| argD | - |
Erwinia amylovora |
General Information
| General Information | Comment | Organism |
|---|---|---|
| physiological function | the argD gene encodes a predicted N-acetylornithine aminotransferase enzyme. A mutant having the Tn5 transposon inserted after nucleotide 999 in the argD gene-coding region, is an arginine auxotroph that does not cause fire blight in apple and has reduced virulence in immature pear fruits. Even when mixed with virulent cells and inoculated onto immature apple fruit, the Tn5 mutant still fails to grow. The ArgD protein cannot be considered an Erwinia amylovora virulence factor because the argD(1000)::Tn5 mutant is auxotrophic and has a primary metabolism defect | Erwinia amylovora |
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