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Literature summary for 2.3.1.261 extracted from
- Attenuation of Mycobacterium tuberculosis by disruption of a mas-like gene or a chalcone synthase-like gene, which causes deficiency in dimycocerosyl phthiocerol synthesis (2003), J. Bacteriol., 185, 2999-3008.
Organism
| Organism | UniProt | Comment | Textmining |
|---|---|---|---|
| Mycobacterium tuberculosis | P96285 | - |
- |
| Mycobacterium tuberculosis H37Rv | P96285 | - |
- |
Synonyms
| Synonyms | Comment | Organism |
|---|---|---|
| msl7 | - |
Mycobacterium tuberculosis |
| PKS1 | - |
Mycobacterium tuberculosis |
| Pks15 | - |
Mycobacterium tuberculosis |
General Information
| General Information | Comment | Organism |
|---|---|---|
| physiological function | a gene disruption mutant is not able to produce dimycocerosyl phthiocerol, although it can produce mycocerosic acids. The mutant is attenuated in a murine model, supporting the hypothesis that dimycocerosyl phthiocerol is a virulence factor | Mycobacterium tuberculosis |
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