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Literature summary for 1.6.1.2 extracted from

  • Leskov, I.; Neville, A.; Shen, X.; Pardue, S.; Kevil, C.G.; Granger, D.N.; Krzywanski, D.M.
    Nicotinamide nucleotide transhydrogenase activity impacts mitochondrial redox balance and the development of hypertension in mice (2017), J. Am. Soc. Hypertens., 11, 110-121 .
    View publication on PubMed

Localization

Localization Comment Organism GeneOntology No. Textmining
mitochondrion
-
Mus musculus 5739
-

Natural Substrates/ Products (Substrates)

Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
NADPH + NAD+ Mus musculus
-
NADP+ + NADH
-
?
NADPH + NAD+ Mus musculus C57/BL6J
-
NADP+ + NADH
-
?

Organism

Organism UniProt Comment Textmining
Mus musculus
-
-
-
Mus musculus C57/BL6J
-
-
-

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
NADPH + NAD+
-
Mus musculus NADP+ + NADH
-
?
NADPH + NAD+
-
Mus musculus C57/BL6J NADP+ + NADH
-
?

Synonyms

Synonyms Comment Organism
nicotinamide nucleotide transhydrogenase
-
Mus musculus
NNT
-
Mus musculus

General Information

General Information Comment Organism
malfunction the absence of enzyme leads to variation in mitochondrial function and contributes to a unique mitochondrial redox phenotype that influences susceptibility to hypertension by contributing to endothelial and vascular dysfunction Mus musculus
physiological function the enzyme activity impacts mitochondrial redox balance and the development of hypertension in mice Mus musculus