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Adenocarcinoma
Fatty acid CoA ligase 4 is up-regulated in colon adenocarcinoma.
Adenocarcinoma of Lung
High-fat diet impairs ferroptosis and promotes cancer invasiveness via downregulating tumor suppressor ACSL4 in lung adenocarcinoma.
Adenoma
Fatty acid CoA ligase 4 is up-regulated in colon adenocarcinoma.
arachidonate-coa ligase deficiency
Liver-specific knockdown of long-chain acyl-CoA synthetase 4 reveals its key role in VLDL-TG metabolism and phospholipid synthesis in mice fed a high-fat diet.
arachidonate-coa ligase deficiency
Long-chain acyl-CoA synthetase 4 participates in the formation of highly unsaturated fatty acid-containing phospholipids in murine macrophages.
arachidonate-coa ligase deficiency
Tissue Specific Ablation of ACSL4 Results in Disturbed Steroidogenesis.
Atherosclerosis
Rosiglitazone inhibits acyl-CoA synthetase activity and fatty acid partitioning to diacylglycerol and triacylglycerol via a peroxisome proliferator-activated receptor-gamma-independent mechanism in human arterial smooth muscle cells and macrophages.
Atherosclerosis
Verification of ferroptosis and pyroptosis and identification of PTGS2 as the hub gene in human coronary artery atherosclerosis.
Brain Edema
Acyl-CoA synthetase long chain family member 4 plays detrimental role in early brain injury after subarachnoid hemorrhage in rats by inducing ferroptosis.
Brain Infarction
Inhibition of Acyl-CoA Synthetase Long-Chain Family Member 4 Facilitates Neurological Recovery After Stroke by Regulation Ferroptosis.
Brain Injuries
ACSL4 exacerbates ischemic stroke by promoting ferroptosis-induced brain injury and neuroinflammation.
Brain Injuries
Acyl-CoA synthetase long chain family member 4 plays detrimental role in early brain injury after subarachnoid hemorrhage in rats by inducing ferroptosis.
Brain Ischemia
ACSL4 exacerbates ischemic stroke by promoting ferroptosis-induced brain injury and neuroinflammation.
Breast Neoplasms
17?-estradiol-induced ACSL4 protein expression promotes an invasive phenotype in estrogen receptor positive mammary carcinoma cells.
Breast Neoplasms
ACSL4 dictates ferroptosis sensitivity by shaping cellular lipid composition.
Breast Neoplasms
Acyl-CoA synthetase-4 mediates radioresistance of breast cancer cells by regulating FOXM1.
Breast Neoplasms
Acyl-CoA synthetase-4, a new regulator of mTOR and a potential therapeutic target for enhanced estrogen receptor function in receptor-positive and -negative breast cancer.
Breast Neoplasms
An Evaluation of Fatty Acid-CoA Ligase 4 in Breast Cancer.
Breast Neoplasms
Association of long-chain acyl-coenzyme A synthetase 5 expression in human breast cancer by estrogen receptor status and its clinical significance.
Breast Neoplasms
Expression of Long-chain Fatty Acyl-CoA Synthetase 4 in Breast and Prostate Cancers Is Associated with Sex Steroid Hormone Receptor Negativity.
Breast Neoplasms
Functional interaction between acyl-CoA synthetase 4, lipooxygenases and cyclooxygenase-2 in the aggressive phenotype of breast cancer cells.
Breast Neoplasms
Long chain fatty Acyl-CoA synthetase 4 is a biomarker for and mediator of hormone resistance in human breast cancer.
Breast Neoplasms
PADI2 gene confers susceptibility to breast cancer and plays tumorigenic role via ACSL4, BINC3 and CA9 signaling.
Breast Neoplasms
Polyphyllin ?-Induced Ferroptosis in MDA-MB-231 Triple-Negative Breast Cancer Cells can Be Protected Against by KLF4-Mediated Upregulation of xCT.
Breast Neoplasms
Predictive and prognostic impact of ferroptosis-related genes ACSL4 and GPX4 on breast cancer treated with neoadjuvant chemotherapy.
Breast Neoplasms
Regulatory mechanisms leading to differential Acyl-CoA synthetase 4 expression in breast cancer cells.
Breast Neoplasms
The endogenous subcellular localisations of the long chain fatty acid-activating enzymes ACSL3 and ACSL4 in sarcoma and breast cancer cells.
Breast Neoplasms
The functional interaction between Acyl-CoA synthetase 4, 5-lipooxygenase and cyclooxygenase-2 controls tumor growth: a novel therapeutic target.
Breast Neoplasms
Tyrosine phosphatase SHP2 regulates the expression of acyl-CoA synthetase ACSL4.
Carcinogenesis
Fatty acid CoA ligase 4 is up-regulated in colon adenocarcinoma.
Carcinogenesis
Fatty acid-CoA ligase 4 is overexpressed in human hepatocellular carcinoma.
Carcinogenesis
Functional interaction between acyl-CoA synthetase 4, lipooxygenases and cyclooxygenase-2 in the aggressive phenotype of breast cancer cells.
Carcinogenesis
Intracellular unesterified arachidonic acid signals apoptosis.
Carcinogenesis
Involvement of fatty acid-CoA ligase 4 in hepatocellular carcinoma growth: roles of cyclic AMP and p38 mitogen-activated protein kinase.
Carcinogenesis
PADI2 gene confers susceptibility to breast cancer and plays tumorigenic role via ACSL4, BINC3 and CA9 signaling.
Carcinogenesis
Regulation of cell growth by fatty acid-CoA ligase 4 in human hepatocellular carcinoma cells.
Carcinogenesis
The functional interaction between Acyl-CoA synthetase 4, 5-lipooxygenase and cyclooxygenase-2 controls tumor growth: a novel therapeutic target.
Carcinogenesis
The positive feedback between ACSL4 expression and O-GlcNAcylation contributes to the growth and survival of hepatocellular carcinoma.
Carcinogenesis
Tyrosine phosphatase SHP2 regulates the expression of acyl-CoA synthetase ACSL4.
Carcinoma
17?-estradiol-induced ACSL4 protein expression promotes an invasive phenotype in estrogen receptor positive mammary carcinoma cells.
Carcinoma
Oleanolic acid inhibits cervical cancer Hela cell proliferation through modulation of the ACSL4 ferroptosis signaling pathway.
Carcinoma
The effect of fatty acid-CoA ligase 4 on the growth of hepatic cancer cells.
Carcinoma, Hepatocellular
ACSL4 is a predictive biomarker of sorafenib sensitivity in hepatocellular carcinoma.
Carcinoma, Hepatocellular
ACSL4 promotes hepatocellular carcinoma progression via c-Myc stability mediated by ERK/FBW7/c-Myc axis.
Carcinoma, Hepatocellular
ACSL4 reprograms fatty acid metabolism in hepatocellular carcinoma via c-Myc/SREBP1 pathway.
Carcinoma, Hepatocellular
Fatty acid-CoA ligase 4 is overexpressed in human hepatocellular carcinoma.
Carcinoma, Hepatocellular
Human MicroRNA-548p Decreases Hepatic Apolipoprotein B Secretion and Lipid Synthesis.
Carcinoma, Hepatocellular
Identification of MiR-211-5p as a tumor suppressor by targeting ACSL4 in Hepatocellular Carcinoma.
Carcinoma, Hepatocellular
Immunohistochemical staining reveals differential expression of ACSL3 and ACSL4 in hepatocellular carcinoma and hepatic gastrointestinal metastases.
Carcinoma, Hepatocellular
Involvement of cholesterol in hepatitis B virus X protein-induced abnormal lipid metabolism of hepatoma cells via up-regulating miR-205-targeted ACSL4.
Carcinoma, Hepatocellular
Involvement of fatty acid-CoA ligase 4 in hepatocellular carcinoma growth: roles of cyclic AMP and p38 mitogen-activated protein kinase.
Carcinoma, Hepatocellular
Overexpression of Acyl-CoA Ligase 4 (ACSL4) in Patients with Hepatocellular Carcinoma and its Prognosis.
Carcinoma, Hepatocellular
Regulation of cell growth by fatty acid-CoA ligase 4 in human hepatocellular carcinoma cells.
Carcinoma, Hepatocellular
Signature molecules expressed differentially in a liver disease stage-specific manner by HIV-1 and HCV co-infection.
Carcinoma, Hepatocellular
Simultaneous silencing of ACSL4 and induction of GADD45B in hepatocellular carcinoma cells amplifies the synergistic therapeutic effect of aspirin and sorafenib.
Carcinoma, Hepatocellular
The positive feedback between ACSL4 expression and O-GlcNAcylation contributes to the growth and survival of hepatocellular carcinoma.
Cholangiocarcinoma
Immunohistochemical staining reveals differential expression of ACSL3 and ACSL4 in hepatocellular carcinoma and hepatic gastrointestinal metastases.
Colonic Neoplasms
Cytoglobin promotes sensitivity to ferroptosis by regulating p53-YAP1 axis in colon cancer cells.
Colonic Neoplasms
Fatty acid CoA ligase 4 is up-regulated in colon adenocarcinoma.
Colonic Neoplasms
Fatty acid-CoA ligase 4 is overexpressed in human hepatocellular carcinoma.
Colorectal Neoplasms
Systematic Analysis of Gene Expression Alterations and Clinical Outcomes for Long-Chain Acyl-Coenzyme A Synthetase Family in Cancer.
Epilepsy
Array-CGH in unclear syndromic nephropathies identifies a microdeletion in Xq22.3-q23.
Erythema
Association of a long-chain fatty acid-CoA ligase 4 gene polymorphism with depression and with enhanced niacin-induced dermal erythema.
Fatty Liver
Comparative transcriptomic analysis reveals an association of gibel carp fatty liver with ferroptosis pathway.
Fatty Liver
Identification of p115 as a novel ACSL4 interacting protein and its role in regulating ACSL4 degradation.
Fatty Liver
Therapeutic Targeting of Hepatic ACSL4 Ameliorates Non-alcoholic Steatohepatitis in Mice.
Fibrosarcoma
The endogenous subcellular localisations of the long chain fatty acid-activating enzymes ACSL3 and ACSL4 in sarcoma and breast cancer cells.
Fibrosarcoma
Triacsin C: a differential inhibitor of arachidonoyl-CoA synthetase and nonspecific long chain acyl-CoA synthetase.
Glioblastoma
Dihydroartemisinin initiates ferroptosis in glioblastoma through GPX4 inhibition.
Glioblastoma
MicroRNA-670-3p suppresses ferroptosis of human glioblastoma cells through targeting ACSL4.
Glioma
ACSL4 suppresses glioma cells proliferation via activating ferroptosis.
Glioma
Integrated metabolomics and lipidomics analyses reveal metabolic reprogramming in human glioma with IDH1 mutation.
Glioma
Regulation and role of Acyl-CoA synthetase 4 in glial cells.
Hemoglobinuria
Induction of two independent immunological cell death signaling following hemoglobinuria -induced acute kidney injury: In vivo study.
Hepatitis B
Involvement of cholesterol in hepatitis B virus X protein-induced abnormal lipid metabolism of hepatoma cells via up-regulating miR-205-targeted ACSL4.
Hypersensitivity
Inhibition of ferroptosis-like cell death attenuates neuropathic pain reactions induced by peripheral nerve injury in rats.
Infarction, Middle Cerebral Artery
Inhibition of Acyl-CoA Synthetase Long-Chain Family Member 4 Facilitates Neurological Recovery After Stroke by Regulation Ferroptosis.
Intellectual Disability
A novel 47.2 Mb duplication on chromosomal bands Xq21.1-25 associated with mental retardation.
Intellectual Disability
A third MRX family (MRX68) is the result of mutation in the long chain fatty acid-CoA ligase 4 (FACL4) gene: proposal of a rapid enzymatic assay for screening mentally retarded patients.
Intellectual Disability
dAcsl, the Drosophila ortholog of acyl-CoA synthetase long-chain family member 3 and 4, inhibits synapse growth by attenuating bone morphogenetic protein signaling via endocytic recycling.
Intellectual Disability
Decreased expression of genes associated with memory and x-linked mental retardation in boys with non-syndromic cryptorchidism and high infertility risk.
Intellectual Disability
FACL4, a new gene encoding long-chain acyl-CoA synthetase 4, is deleted in a family with Alport syndrome, elliptocytosis, and mental retardation.
Intellectual Disability
FACL4, encoding fatty acid-CoA ligase 4, is mutated in nonspecific X-linked mental retardation.
Intellectual Disability
Identification and characterization of mouse orthologs of the AMMECR1 and FACL4 genes deleted in AMME syndrome: orthology of Xq22.3 and MmuXF1-F3.
Intellectual Disability
Intellectual disability, midface hypoplasia, facial hypotonia, and Alport syndrome are associated with a deletion in Xq22.3.
Intellectual Disability
Localization of a non-syndromic X-linked mental retardation gene (MRX80) to Xq22-q24.
Intellectual Disability
No association between polymorphisms in the FACL4 (fatty acid-CoA ligase 4) gene and nonspecific mental retardation in Qin-Ba mountain region of China.
Intellectual Disability
The XLMR gene ACSL4 plays a role in dendritic spine architecture.
Intellectual Disability
X linked mental retardation: a clinical guide.
Intellectual Disability
Xq22.3-q23 deletion including ACSL4 in a patient with intellectual disability.
Ischemic Attack, Transient
Inhibition of Acyl-CoA Synthetase Long-Chain Family Member 4 Facilitates Neurological Recovery After Stroke by Regulation Ferroptosis.
Ischemic Stroke
ACSL4 exacerbates ischemic stroke by promoting ferroptosis-induced brain injury and neuroinflammation.
Kidney Diseases
XJB-5-131 inhibited ferroptosis in tubular epithelial cells after ischemia-reperfusion injury.
Leiomyosarcoma
The endogenous subcellular localisations of the long chain fatty acid-activating enzymes ACSL3 and ACSL4 in sarcoma and breast cancer cells.
Lissencephaly
Array-CGH in unclear syndromic nephropathies identifies a microdeletion in Xq22.3-q23.
Liver Diseases
Identification of p115 as a novel ACSL4 interacting protein and its role in regulating ACSL4 degradation.
Liver Diseases
Signature molecules expressed differentially in a liver disease stage-specific manner by HIV-1 and HCV co-infection.
Liver Neoplasms
A Novel TIP30 Protein Complex Regulates EGF Receptor Signaling and Endocytic Degradation.
Liver Neoplasms
The effect of fatty acid-CoA ligase 4 on the growth of hepatic cancer cells.
Lung Injury
Inhibition of ACSL4 attenuates ferroptotic damage after pulmonary ischemia-reperfusion.
Lung Neoplasms
Systematic Analysis of Gene Expression Alterations and Clinical Outcomes for Long-Chain Acyl-Coenzyme A Synthetase Family in Cancer.
Mental Retardation, X-Linked
A third MRX family (MRX68) is the result of mutation in the long chain fatty acid-CoA ligase 4 (FACL4) gene: proposal of a rapid enzymatic assay for screening mentally retarded patients.
Mental Retardation, X-Linked
Analyses of mental dysfunction-related ACSl4 in Drosophila reveal its requirement for Dpp/BMP production and visual wiring in the brain.
Mental Retardation, X-Linked
Drosophila Acyl-CoA synthetase long-chain family member 4 regulates axonal transport of synaptic vesicles and is required for synaptic development and transmission.
Mental Retardation, X-Linked
FACL4, encoding fatty acid-CoA ligase 4, is mutated in nonspecific X-linked mental retardation.
Mental Retardation, X-Linked
The XLMR gene ACSL4 plays a role in dendritic spine architecture.
Mental Retardation, X-Linked
[Monogenic causes of nonspecific X-linked mental retardation molecular aspects]
Mental Retardation, X-Linked
[Relationship between FACL4 and non-specific X-linked mental retardation]
Mouth Neoplasms
Non-classical platinum-based compound 56MESS, with preferential cytotoxic effect on oral cancer cells by downregulating FACL4 expression.
Muscle Hypotonia
Disruption of DMD and deletion of ACSL4 causing developmental delay, hypotonia, and multiple congenital anomalies.
Muscle Hypotonia
Intellectual disability, midface hypoplasia, facial hypotonia, and Alport syndrome are associated with a deletion in Xq22.3.
Neoplasm Metastasis
ACSL4 reprograms fatty acid metabolism in hepatocellular carcinoma via c-Myc/SREBP1 pathway.
Neoplasm Metastasis
Immunohistochemical staining reveals differential expression of ACSL3 and ACSL4 in hepatocellular carcinoma and hepatic gastrointestinal metastases.
Neoplasms
ACSL family: The regulatory mechanisms and therapeutic implications in cancer.
Neoplasms
ACSL4 is a predictive biomarker of sorafenib sensitivity in hepatocellular carcinoma.
Neoplasms
ACSL4 promotes hepatocellular carcinoma progression via c-Myc stability mediated by ERK/FBW7/c-Myc axis.
Neoplasms
Acyl-CoA synthetase-4 is implicated in drug resistance in breast cancer cell lines involving the regulation of energy-dependent transporter expression.
Neoplasms
Acyl-CoA synthetase-4, a new regulator of mTOR and a potential therapeutic target for enhanced estrogen receptor function in receptor-positive and -negative breast cancer.
Neoplasms
Evaluation of long-chain acyl-coenzyme A synthetase 4 (ACSL4) expression in human breast cancer.
Neoplasms
Fatty acid activation in carcinogenesis and cancer development: Essential roles of long-chain acyl-CoA synthetases.
Neoplasms
Fatty acid CoA ligase 4 is up-regulated in colon adenocarcinoma.
Neoplasms
Fatty acid-CoA ligase 4 is overexpressed in human hepatocellular carcinoma.
Neoplasms
High-fat diet impairs ferroptosis and promotes cancer invasiveness via downregulating tumor suppressor ACSL4 in lung adenocarcinoma.
Neoplasms
Identification of MiR-211-5p as a tumor suppressor by targeting ACSL4 in Hepatocellular Carcinoma.
Neoplasms
Immunohistochemical staining reveals differential expression of ACSL3 and ACSL4 in hepatocellular carcinoma and hepatic gastrointestinal metastases.
Neoplasms
Long chain fatty Acyl-CoA synthetase 4 is a biomarker for and mediator of hormone resistance in human breast cancer.
Neoplasms
Long-Chain Acyl-CoA Synthetase 4-Mediated Fatty Acid Metabolism Sustains Androgen Receptor Pathway-Independent Prostate Cancer.
Neoplasms
New inhibitor targeting Acyl-CoA synthetase 4 reduces breast and prostate tumor growth, therapeutic resistance and steroidogenesis.
Neoplasms
Overexpression of Acyl-CoA Ligase 4 (ACSL4) in Patients with Hepatocellular Carcinoma and its Prognosis.
Neoplasms
PADI2 gene confers susceptibility to breast cancer and plays tumorigenic role via ACSL4, BINC3 and CA9 signaling.
Neoplasms
Predictive and prognostic impact of ferroptosis-related genes ACSL4 and GPX4 on breast cancer treated with neoadjuvant chemotherapy.
Neoplasms
Role of acyl-CoA synthetase ACSL4 in arachidonic acid metabolism.
Neoplasms
The effect of fatty acid-CoA ligase 4 on the growth of hepatic cancer cells.
Neoplasms
The endogenous subcellular localisations of the long chain fatty acid-activating enzymes ACSL3 and ACSL4 in sarcoma and breast cancer cells.
Neoplasms
The functional interaction between Acyl-CoA synthetase 4, 5-lipooxygenase and cyclooxygenase-2 controls tumor growth: a novel therapeutic target.
Neoplasms
Tumor suppressor miR-424-5p abrogates ferroptosis in ovarian cancer through targeting ACSL4.
Neoplasms
Tumor-suppressive functions of long-chain acyl-CoA synthetase 4 in gastric cancer.
Nephritis, Hereditary
Array-CGH in unclear syndromic nephropathies identifies a microdeletion in Xq22.3-q23.
Nephritis, Hereditary
FACL4, a new gene encoding long-chain acyl-CoA synthetase 4, is deleted in a family with Alport syndrome, elliptocytosis, and mental retardation.
Nephritis, Hereditary
Identification and characterization of a highly conserved protein absent in the Alport syndrome (A), mental retardation (M), midface hypoplasia (M), and elliptocytosis (E) contiguous gene deletion syndrome (AMME).
Nephritis, Hereditary
Identification and characterization of mouse orthologs of the AMMECR1 and FACL4 genes deleted in AMME syndrome: orthology of Xq22.3 and MmuXF1-F3.
Nephritis, Hereditary
Intellectual disability, midface hypoplasia, facial hypotonia, and Alport syndrome are associated with a deletion in Xq22.3.
Neuroinflammatory Diseases
ACSL4 exacerbates ischemic stroke by promoting ferroptosis-induced brain injury and neuroinflammation.
Neuroinflammatory Diseases
Tetrahydroxy stilbene glycoside ameliorates Alzheimer's disease in APP/PS1 mice via glutathione peroxidase related ferroptosis.
Neuromuscular Diseases
Disruption of DMD and deletion of ACSL4 causing developmental delay, hypotonia, and multiple congenital anomalies.
Non-alcoholic Fatty Liver Disease
Genome-wide analysis of DNA methylation in human peripheral leukocytes identifies potential biomarkers of nonalcoholic fatty liver disease.
Non-alcoholic Fatty Liver Disease
Hepatic gene expression of Caucasian and African-American patients with obesity-related non-alcoholic fatty liver disease.
Non-alcoholic Fatty Liver Disease
Identification of p115 as a novel ACSL4 interacting protein and its role in regulating ACSL4 degradation.
Non-alcoholic Fatty Liver Disease
Therapeutic Targeting of Hepatic ACSL4 Ameliorates Non-alcoholic Steatohepatitis in Mice.
Obesity
Genes involved in fatty acid partitioning and binding, lipolysis, monocyte/macrophage recruitment, and inflammation are overexpressed in the human fatty liver of insulin-resistant subjects.
Ovarian Neoplasms
Systems biology of ferroptosis: A modeling approach.
Ovarian Neoplasms
Tumor suppressor miR-424-5p abrogates ferroptosis in ovarian cancer through targeting ACSL4.
Pancreatic Neoplasms
Identification of prognostic lipid droplet-associated genes in pancreatic cancer patients via bioinformatics analysis.
Prostatic Neoplasms
ACSL4 promotes prostate cancer growth, invasion and hormonal resistance.
Prostatic Neoplasms
Acyl-CoA synthetase-4 is implicated in drug resistance in breast cancer cell lines involving the regulation of energy-dependent transporter expression.
Prostatic Neoplasms
Expression of Long-chain Fatty Acyl-CoA Synthetase 4 in Breast and Prostate Cancers Is Associated with Sex Steroid Hormone Receptor Negativity.
Prostatic Neoplasms
LncRNA NEAT1 promotes docetaxel resistance in prostate cancer by regulating ACSL4 via sponging miR-34a-5p and miR-204-5p.
Prostatic Neoplasms
Long-Chain Acyl-CoA Synthetase 4-Mediated Fatty Acid Metabolism Sustains Androgen Receptor Pathway-Independent Prostate Cancer.
Rhabdomyosarcoma
The endogenous subcellular localisations of the long chain fatty acid-activating enzymes ACSL3 and ACSL4 in sarcoma and breast cancer cells.
Sarcoma
The endogenous subcellular localisations of the long chain fatty acid-activating enzymes ACSL3 and ACSL4 in sarcoma and breast cancer cells.
Sepsis
Long-Chain Acyl-CoA Synthetase 1 Role in Sepsis and Immunity: Perspectives From a Parallel Review of Public Transcriptome Datasets and of the Literature.
Stomach Neoplasms
Tumor-suppressive functions of long-chain acyl-CoA synthetase 4 in gastric cancer.
Stroke
An ACSL4 Hemizygous Intragenic Deletion in a Patient With Childhood Stroke.
Stroke
Inhibition of Acyl-CoA Synthetase Long-Chain Family Member 4 Facilitates Neurological Recovery After Stroke by Regulation Ferroptosis.
Subarachnoid Hemorrhage
Acyl-CoA synthetase long chain family member 4 plays detrimental role in early brain injury after subarachnoid hemorrhage in rats by inducing ferroptosis.
Triple Negative Breast Neoplasms
Long chain fatty Acyl-CoA synthetase 4 is a biomarker for and mediator of hormone resistance in human breast cancer.
Triple Negative Breast Neoplasms
Regulatory mechanisms leading to differential Acyl-CoA synthetase 4 expression in breast cancer cells.
Uterine Cervical Neoplasms
Oleanolic acid inhibits cervical cancer Hela cell proliferation through modulation of the ACSL4 ferroptosis signaling pathway.
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