Reference on EC 3.5.1.109 - sphingomyelin deacylase

Please use the Reference Search for a specific query.

Please wait a moment until all data is loaded. This message will disappear when all data is loaded.

Higuchi, K.; Hara, J.; Okamoto, R.; Kawashima, M.; Imokawa, G.

The skin of atopic dermatitis patients contains a novel enzyme, glucosylceramide sphingomyelin deacylase, which cleaves the N-acyl linkage of sphingomyelin and glucosylceramide

Biochem. J.

350

747-756

2000

Homo sapiens

10970788

712591

Imokawa, G.

A possible mechanism underlying the ceramide deficiency in atopic dermatitis: expression of a deacylase enzyme that cleaves the N-acyl linkage of sphingomyelin and glucosylceramide

J. Dermatol. Sci.

1-9

2009

Homo sapiens

19443184

712668

Hara, J.; Higuchi, K.; Okamoto, R.; Kawashima, M.; Imokawa, G.

High-expression of sphingomyelin deacylase is an important determinant of ceramide deficiency leading to barrier disruption in atopic dermatitis

J. Invest. Dermatol.

115

406-413

2000

Homo sapiens

10951276

712912

Ishibashi, M.; Arikawa, J.; Okamoto, R.; Kawashima, M.; Takagi, Y.; Ohguchi, K.; Imokawa, G.

Abnormal expression of the novel epidermal enzyme, glucosylceramide deacylase, and the accumulation of its enzymatic reaction product, glucosylsphingosine, in the skin of patients with atopic dermatitis

Lab. Invest.

397-408

2003

Homo sapiens

12649340