identification of the lenc1, i.e. low expression of NCED3 1, mutation, the mutant shows lower AtNCED3 promoter activity after dehydration treatment compared to the wild-type, the lenc1 mutation is recessive and is located on chromosome 4, phenotype, overview. The lenc1 mutant is hypersensitive to methyl viologen, LiCl, NaCl, and high light. The aerial part of lenc1 looses water faster than wild-type possibly due to a larger stomata opening
identification, expression analysis and phenotype of the lenc1 mutant, i.e. low expression of NCED3 1, mutation, developmental phenotypes of lenc1 mutant, overview. The mutant shows lower AtNCED3 promoter activity after dehydration treatment compared to the wild-type, the lenc1 mutation is recessive and is located on chromosome 4, phenotype, overview. The lenc1 mutant is hypersensitive to methyl viologen, LiCl, NaCl, and high light. The aerial part of lenc1 looses water faster than wild-type possibly due to a larger stomata opening
induction of 9-cis-epoxycarotenoid dioxygenase in Arabidopsis thaliana seeds enhances seed dormancy. Development of transgenic plants, which permit conditional expression of gene NCED6 using the ecdysone receptor-based plant gene switch system and the ligand methoxyfenozide. Induction of NCED6 during imbibition increased ABA levels more than 20fold and is sufficient to prevent seed germination
identification of the lenc1, i.e. low expression of NCED3 1, mutation, the mutant shows lower AtNCED3 promoter activity after dehydration treatment compared to the wild-type, the lenc1 mutation is recessive and is located on chromosome 4, phenotype, overview. The lenc1 mutant is hypersensitive to methyl viologen, LiCl, NaCl, and high light. The aerial part of lenc1 looses water faster than wild-type possibly due to a larger stomata opening
identification, expression analysis and phenotype of the lenc1 mutant, i.e. low expression of NCED3 1, mutation, developmental phenotypes of lenc1 mutant, overview. The mutant shows lower AtNCED3 promoter activity after dehydration treatment compared to the wild-type, the lenc1 mutation is recessive and is located on chromosome 4, phenotype, overview. The lenc1 mutant is hypersensitive to methyl viologen, LiCl, NaCl, and high light. The aerial part of lenc1 looses water faster than wild-type possibly due to a larger stomata opening
identification of the lenc1, i.e. low expression of NCED3 1, mutation, the mutant shows lower AtNCED3 promoter activity after dehydration treatment compared to the wild-type, the lenc1 mutation is recessive and is located on chromosome 4, phenotype, overview. The lenc1 mutant is hypersensitive to methyl viologen, LiCl, NaCl, and high light. The aerial part of lenc1 looses water faster than wild-type possibly due to a larger stomata opening
identification, expression analysis and phenotype of the lenc1 mutant, i.e. low expression of NCED3 1, mutation, developmental phenotypes of lenc1 mutant, overview. The mutant shows lower AtNCED3 promoter activity after dehydration treatment compared to the wild-type, the lenc1 mutation is recessive and is located on chromosome 4, phenotype, overview. The lenc1 mutant is hypersensitive to methyl viologen, LiCl, NaCl, and high light. The aerial part of lenc1 looses water faster than wild-type possibly due to a larger stomata opening
induction of 9-cis-epoxycarotenoid dioxygenase in Arabidopsis thaliana seeds enhances seed dormancy. Development of transgenic plants, which permit conditional expression of gene NCED6 using the ecdysone receptor-based plant gene switch system and the ligand methoxyfenozide. Induction of NCED6 during imbibition increased ABA levels more than 20fold and is sufficient to prevent seed germination
fruit-specific suppression of gene SlNCED1, avoid the pleiotropic phenotypes associated with abscisic acid deficiency, using fruit-specific E8 promoter in RNAi constructs, abscisic acid accumulation and SlNCED1 transcript levels in the transgenic fruit are downregulated to between 20% and 50% of the levels measured in the control fruit. Significant reduction in 9-cis-epoxycarotenoid dioxygenase activity leads to a downregulation in the transcription of genes encoding major cell wall catabolic enzymes, specifically polygalacturonase, pectin methyl esterase, beta-galactosidase precursor mRNA, xyloglucan endotransglycosylase, endo-1,4-beta cellulose, and expansin resulting in an increased accumulation of pectin during ripening and thus to significant extension of the shelf life to 15 to 29 d compared with a shelf life of only 7 d for the control fruit and an enhancement of fruit firmness at the mature stage by 30% to 45%