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Literature summary for 4.2.1.146 extracted from

  • Zhang, L.; Thiewes, H.; van Kan, J.A.
    The D-galacturonic acid catabolic pathway in Botrytis cinerea (2011), Fungal Genet. Biol., 48, 990-997.
    View publication on PubMed
Search for more literature for 4.2.1.146

Cloned(Commentary)

Cloned (Comment) Organism
gene Bclgd1, quantitative RT-PCR expression analysis Botrytis cinerea

Protein Variants

Protein Variants Comment Organism
additional information gene replacement strategy for generating Bortrytis cinerea deletion constructs, complementation study, overview. Targeted gene replacement of all four genes, either separately or in combinations, yields mutants that are affected in growth on D-galacturonic acid, pectate, or pectin as the sole carbon source Botrytis cinerea

Organism

Organism UniProt Comment Textmining
Botrytis cinerea
-
 gene Bclgd1
-

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
L-galactonate
-
 Botrytis cinerea 2-keto-3-deoxy-L-galactonate + H2O
-
 ?
L-galactonate
-
 Botrytis cinerea B05.10 2-keto-3-deoxy-L-galactonate + H2O
-
 ?

Synonyms

Synonyms Comment Organism
LGD1
-
 Botrytis cinerea

Temperature Optimum [°C]

Temperature Optimum [°C] Temperature Optimum Maximum [°C] Comment Organism
22
-
 assay at room temperature Botrytis cinerea

pH Optimum

pH Optimum Minimum pH Optimum Maximum Comment Organism
5.2
-
 assay at Botrytis cinerea

Expression

Organism Comment Expression
Botrytis cinerea pectate, pectinate, and galacturonic acid suppress enmzyme expression down
Botrytis cinerea glucose induces the enzyme up

General Information

General Information Comment Organism
metabolism the enzyme catalyzes the second step in the D-galacturonic acid catabolic pathway of the fungus Botrytis cinerea
additional information the virulence of all Bortrytis cinerea mutants in the D-galacturonic acid catabolic pathway on tomato leaves, apple fruit and bell peppers is unaltered Botrytis cinerea