Literature summary for 3.4.21.5 extracted from

Alexandru, N.; Constantin, A.; Popov, D.
Carbonylation of platelet proteins occurs as consequence of oxidative stress and thrombin activation, and is stimulated by ageing and type 2 diabetes (2008), Clin. Chem. Lab. Med., 46, 528-536.

Search for more literature for 3.4.21.5

Application

Application	Comment	Organism
medicine	exogenous oxidative stress, thrombin activation, progression of ageing and type 2 diabetes lead to protein carbonyls formation in platelets, and this modification can be attenuated by antioxidant enzymes	Homo sapiens
medicine	exogenous oxidative stress, thrombin activation, progression of ageing and type 2 diabetes lead to protein carbonyls formation in platelets, and this modification can be attenuated by antioxidant enzymes	Rattus norvegicus
medicine	exogenous oxidative stress, thrombin activation, progression of ageing and type 2 diabetes lead to protein carbonyls formation in platelets, and this modification can be attenuated by antioxidant enzymes	Mesocricetus auratus

Organism

Organism	UniProt	Comment	Textmining
Homo sapiens	-	-	-
Mesocricetus auratus	-	-	-
Rattus norvegicus	-	-	-

Source Tissue

Source Tissue	Comment	Organism	Textmining
blood platelet	exogenous oxidative stress, thrombin activation, progression of ageing and type 2 diabetes lead to protein carbonyls formation in platelets, and this modification can be attenuated by antioxidant enzymes	Homo sapiens	-
blood platelet	exogenous oxidative stress, thrombin activation, progression of ageing and type 2 diabetes lead to protein carbonyls formation in platelets, and this modification can be attenuated by antioxidant enzymes	Rattus norvegicus	-
blood platelet	exogenous oxidative stress, thrombin activation, progression of ageing and type 2 diabetes lead to protein carbonyls formation in platelets, and this modification can be attenuated by antioxidant enzymes	Mesocricetus auratus	-

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Release 2023.1 (January 2023)