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Literature summary for 3.2.1.39 extracted from
- Silencing of acidic pathogenesis-related PR-1 genes increases extracellular beta-(1-3)-glucanase activity at the onset of tobacco defence reactions (2008), J. Exp. Bot., 59, 1225-1239.
Activating Compound
| Activating Compound | Comment | Organism | Structure |
|---|---|---|---|
| additional information | PR-1 silencing specifically increases beta-(1-3)-glucanase activity, suggesting a role for acidic PR-1 in the negative regulation of beta-(1-3)-glucanase activity | Nicotiana tabacum |
Cloned(Commentary)
| Cloned (Comment) | Organism |
|---|---|
| expression in Pichia pastoris | Nicotiana tabacum |
Organism
| Organism | UniProt | Comment | Textmining |
|---|---|---|---|
| Nicotiana tabacum | - |
cv. Xanthi nc. | - |
Purification (Commentary)
| Purification (Comment) | Organism |
|---|---|
- |
Nicotiana tabacum |
Substrates and Products (Substrate)
| Substrates | Comment Substrates | Organism | Products | Comment (Products) | Rev. | Reac. |
|---|---|---|---|---|---|---|
| laminarin + H2O | - |
Nicotiana tabacum | ? | - |
? |  |
Synonyms
| Synonyms | Comment | Organism |
|---|---|---|
| beta-1,3-glucanase | - |
Nicotiana tabacum |
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Release 2023.1 (January 2023)
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