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Literature summary for 2.3.1.15 extracted from

  • Linden, D.; William-Olsson, L.; Ahnmark, A.; Ekroos, K.; Hallberg, C.; Sjoegren, H.P.; Becker, B.; Svensson, L.; Clapham, J.C.; Oscarsson, J.; Schreyer, S.
    Liver-directed overexpression of mitochondrial glycerol-3-phosphate acyltransferase results in hepatic steatosis, increased triacylglycerol secretion and reduced fatty acid oxidation (2006), FASEB J., 20, 434-443.
    View publication on PubMed

Cloned(Commentary)

Cloned (Comment) Organism
mice are transduced with adenoviruses expressing either murine mtGPAT or a catalytically inactive variant of the enzyme. Overexpressing mtGPAT results in massive 12- and 7-fold accumulation of liver triacylglycerol and diacylglycerol Mus musculus

Localization

Localization Comment Organism GeneOntology No. Textmining
mitochondrion
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Mus musculus 5739
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Organism

Organism UniProt Comment Textmining
Mus musculus Q61586 precursor
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Source Tissue

Source Tissue Comment Organism Textmining
liver hypothesis: increased hepatic mtGPAT activity associated with obesity and insulin resistance contributes to increased triacylglycerol biosynthesis and inhibition of fatty acid oxidation, responses that would promote hepatic steatosis and dyslipidemia Mus musculus
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Synonyms

Synonyms Comment Organism
mtGPAT
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Mus musculus