The enzyme from rabbit muscle displays absolute stereoselectivity for the beta-anomer of D-fructofuranose 6-phosphate [9-11]. D-Tagatose 6-phosphate and sedoheptulose 7-phosphate can act as acceptors. UTP, CTP and ITP can act as donors. Not identical with EC 2.7.1.105 6-phosphofructo-2-kinase.
The taxonomic range for the selected organisms is: Streptomyces coelicolor The expected taxonomic range for this enzyme is: Bacteria, Eukaryota, Archaea
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SYSTEMATIC NAME
IUBMB Comments
ATP:D-fructose-6-phosphate 1-phosphotransferase
The enzyme from rabbit muscle displays absolute stereoselectivity for the beta-anomer of D-fructofuranose 6-phosphate [9-11]. D-Tagatose 6-phosphate and sedoheptulose 7-phosphate can act as acceptors. UTP, CTP and ITP can act as donors. Not identical with EC 2.7.1.105 6-phosphofructo-2-kinase.
integrated analysis of gene expression data by genome-scale metabolic model simulations, deletion mutant strain produces antibiotics in quantities comparable with that of the reference strain, wild type-like phosphofructokinase activities measured, attributed to intact copy of pfkA2 gene
integrated analysis of gene expression data by genome-scale metabolic model simulations, deletion mutant strain produces antibiotics in quantities comparable with that of the reference strain, wild type-like phosphofructokinase activities measured, attributed to intact copy of pfkA2 gene
integrated analysis of gene expression data by genome-scale metabolic model simulations, deletion mutant strain produces antibiotics in quantities comparable with that of the reference strain, wild type-like phosphofructokinase activities measured, attributed to intact copy of pfkA2 gene
integrated analysis of gene expression data by genome-scale metabolic model simulations, deletion mutant strain produces antibiotics in quantities comparable with that of the reference strain, wild type-like phosphofructokinase activities measured, attributed to intact copy of pfkA2 gene
characterization of the PFKA2-deletion strain by metabolic flux analysis and transcription analysis, transcriptional changes in regulatory and membrane proteins as well as in fatty acid metabolism, carbon flux through the pentose phosphate pathway increased, PFKA2 protein involved in determining the carbon flux distribution, production of the pigmented antibiotics actinorhodin and undecylprodigiosin up to 6fold increased in the deletion strain
characterization of the PFKA2-deletion strain by metabolic flux analysis and transcription analysis, transcriptional changes in regulatory and membrane proteins as well as in fatty acid metabolism, carbon flux through the pentose phosphate pathway increased, PFKA2 protein involved in determining the carbon flux distribution, production of the pigmented antibiotics actinorhodin and undecylprodigiosin up to 6fold increased in the deletion strain
characterization of the PFKA2-deletion strain by metabolic flux analysis and transcription analysis, transcriptional changes in regulatory and membrane proteins as well as in fatty acid metabolism, carbon flux through the pentose phosphate pathway increased, PFKA2 protein involved in determining the carbon flux distribution, production of the pigmented antibiotics actinorhodin and undecylprodigiosin up to 6fold increased in the deletion strain
characterization of the PFKA2-deletion strain by metabolic flux analysis and transcription analysis, transcriptional changes in regulatory and membrane proteins as well as in fatty acid metabolism, carbon flux through the pentose phosphate pathway increased, PFKA2 protein involved in determining the carbon flux distribution, production of the pigmented antibiotics actinorhodin and undecylprodigiosin up to 6fold increased in the deletion strain
integrated analysis of gene expression data by genome-scale metabolic model simulations, deletion mutant strain produces antibiotics in quantities comparable with that of the reference strain, enzyme activities like in wild-type attributed to intact copy of pfkA2 gene
integrated analysis of gene expression data by genome-scale metabolic model simulations, deletion mutant strain produces antibiotics in quantities comparable with that of the reference strain, enzyme activities like in wild-type attributed to intact copy of pfkA2 gene
integrated analysis of gene expression data by genome-scale metabolic model simulations, deletion mutant strain produces antibiotics in quantities comparable with that of the reference strain, enzyme activities like in wild-type attributed to intact copy of pfkA2 gene
integrated analysis of gene expression data by genome-scale metabolic model simulations, deletion mutant strain produces antibiotics in quantities comparable with that of the reference strain, enzyme activities like in wild-type attributed to intact copy of pfkA2 gene
PFKA2-deletion strain, constructed by replacement of a chromosomal sequence in a Streptomyces coelicolor cosmid, 2-6fold increased antibiotic production, accumulation of glucose 6-phosphate, changes of intracellular fluxes, no enzyme activity
PFKA2-deletion strain, constructed by replacement of a chromosomal sequence in a Streptomyces coelicolor cosmid, 2-6fold increased antibiotic production, accumulation of glucose 6-phosphate, changes of intracellular fluxes, no enzyme activity
PFKA2-deletion strain, constructed by replacement of a chromosomal sequence in a Streptomyces coelicolor cosmid, 2-6fold increased antibiotic production, accumulation of glucose 6-phosphate, changes of intracellular fluxes, no enzyme activity
PFKA2-deletion strain, constructed by replacement of a chromosomal sequence in a Streptomyces coelicolor cosmid, 2-6fold increased antibiotic production, accumulation of glucose 6-phosphate, changes of intracellular fluxes, no enzyme activity