3.5.1.99: fatty acid amide hydrolase
This is an abbreviated version!
For detailed information about fatty acid amide hydrolase, go to the full flat file.
Word Map on EC 3.5.1.99
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3.5.1.99
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endocannabinoids
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cannabinoids
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pain
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lipase
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agonist
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monoacylglycerol
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2-arachidonoylglycerol
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anxiety
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analgesic
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cannabis
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n-acylethanolamines
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vanilloid
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2-arachidonoyl
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ethanolamide
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palmitoylethanolamide
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carbamate
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rimonabant
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hyperalgesia
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reward
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tone
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nociceptive
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cannabimimetic
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n-arachidonoylethanolamine
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marijuana
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anxiolytic
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nape-pld
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antinociceptive
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oleoylethanolamide
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emotional
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neuropathic
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delta9-tetrahydrocannabinol
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amidase
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amygdalar
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medicine
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catalepsy
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pharmacology
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anti-allodynic
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anxiolytic-like
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monoglyceride
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cannabidiol
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psychoactive
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sleep-inducing
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phytocannabinoids
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anxiety-like
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arachidonyl
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monoacyl
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capsazepine
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aversive
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tetrahydrocannabinol
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drug development
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anti-hyperalgesic
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neuromodulatory
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fluorophosphonate
- 3.5.1.99
-
endocannabinoids
- cannabinoids
- pain
- lipase
- agonist
- monoacylglycerol
- 2-arachidonoylglycerol
-
anxiety
-
analgesic
- cannabis
- n-acylethanolamines
-
vanilloid
-
2-arachidonoyl
- ethanolamide
- palmitoylethanolamide
- carbamate
-
rimonabant
- hyperalgesia
-
reward
-
tone
-
nociceptive
-
cannabimimetic
- n-arachidonoylethanolamine
- marijuana
-
anxiolytic
- nape-pld
-
antinociceptive
- oleoylethanolamide
-
emotional
-
neuropathic
- delta9-tetrahydrocannabinol
- amidase
-
amygdalar
- medicine
- catalepsy
- pharmacology
-
anti-allodynic
-
anxiolytic-like
- monoglyceride
- cannabidiol
-
psychoactive
-
sleep-inducing
-
phytocannabinoids
-
anxiety-like
-
arachidonyl
-
monoacyl
-
capsazepine
-
aversive
- tetrahydrocannabinol
- drug development
-
anti-hyperalgesic
-
neuromodulatory
- fluorophosphonate
Reaction
Synonyms
AAH, anandamide amidohydrolase, AtFAAH, endocannabinoid-degrading enzyme, FA amide hydrolase, FAAH, FAAH-1, FAAH-2, fatty acid amide hydrolase, fatty-acid amide hydrolase, hFAAH, oleamide hydrolase
ECTree
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Source Tissue
Source Tissue on EC 3.5.1.99 - fatty acid amide hydrolase
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role of FAAH in epithelial cells of the choroid plexus may be to control the concentration of oleamide in the cerebrospinal fluid. FAAH may exert an important regulatory role in shaping the duration and magnitude of the sleep-inducing effect of endogenously or exogenously derived oleamide
additional information
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temporal changes in mouse brain fatty acid amide hydrolase activity, determination by ex vivo autoradiography, overview
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primary sensory, FAAH is localized in the soma, in small dorsal root ganglion neurons
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expression of cannabinoid receptor type 1 and fatty acid amide hydrolase throughout the retina, from the foveal pit to the far periphery, and are present in the photoreceptor, outer plexiform, inner nuclear, inner plexiform, and retinal ganglion cell layers, most prominent CB1R and FAAH expression in cone synaptic terminals and in the ganglion cell layer
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mucosa, urothelium, no FAAH immunoreactivity in other structures of the bladder
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full-wall bladder and mucosa, urothelium, no FAAH immunoreactivity in other structures of the bladder
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mucosa, urothelium, no FAAH immunoreactivity in other structures of the bladder
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mucosa, urothelium, no FAAH immunoreactivity in other structures of the bladder
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determination of the expression patterns of the cannabinoid receptor type 1 and the fatty acid amide hydrolase in the nervous system, triple-labeling immunofluorescence and confocal microscopy analysis, overview. Neither CB1R nor FAAH are found in the retinal glia, the Müller cells
additional information
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not in human HeLa epithelioid carcinoma. Human HMC-1 mast cells show FAAH activity only when 5-lipoxygenase activity is inhibited
additional information
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culturing does not induce major changes in FAAH expression in primary sensory neurons