RNAi-induced depletion of CfCDA2 transcripts, CfCDA2 transcript levels are strongly suppressed in 5thand 6th-instar larvae injected with CfCDA2-specific dsRNA, compared to controls, CfCDA2 transcript levels are 20% and 50% those of controls in injected 5th- and 6th-instar larvae, respectively
RNAi-induced depletion of CfCDA2 transcripts, CfCDA2 transcript levels are strongly suppressed in 5thand 6th-instar larvae injected with CfCDA2-specific dsRNA, compared to controls, CfCDA2 transcript levels are 20% and 50% those of controls in injected 5th- and 6th-instar larvae, respectively
RNAi-induced depletion of CfCDA2 transcripts, CfCDA2 transcript levels are strongly suppressed in 5thand 6th-instar larvae injected with CfCDA2-specific dsRNA, compared to controls, CfCDA2 transcript levels are 20% and 50% those of controls in injected 5th- and 6th-instar larvae, respectively
overexpression of the native, but not of mutated, enzyme interferes with the structural integrity of the intraluminal extracellular matrix and causes aberrant tube elongation, phenotype, overview
LmCDA2 expression is knocked down by RNA interference (RNAi), injection of dsRNA against LmCDA2 into 2-day-old fifth instar nymphs. The dsLmCDA2-injected nymphs fail to shed the old cuticle and are trapped within the exuviae until they died. Their mortality is 80%
LmCDA2 expression is knocked down by RNA interference (RNAi), injection of dsRNA against LmCDA2 into 2-day-old fifth instar nymphs. The dsLmCDA2-injected nymphs fail to shed the old cuticle and are trapped within the exuviae until they died. Their mortality is 80%
LmCDA2 expression is knocked down by RNA interference (RNAi), injection of dsRNA against LmCDA2 into 2-day-old fifth instar nymphs. The dsLmCDA2-injected nymphs fail to shed the old cuticle and are trapped within the exuviae until they died. Their mortality is 80%
construction of the homothallic DELTAcda1 strain by homologous, the mutant forms a little amount of abnormal spores that contain one, two, or three asci, similar to, but not as strong as, the phenotype observed in a deletion mutant of chs1 encoding chitin synthase 1, spore morphology and sporulation in the disruption mutants, overview, the phenotype is reversed by expression of Sacharomyces cerevisiae chitin deacetylase CDA1
construction of the homothallic DELTAcda1 strain by homologous, the mutant forms a little amount of abnormal spores that contain one, two, or three asci, similar to, but not as strong as, the phenotype observed in a deletion mutant of chs1 encoding chitin synthase 1, spore morphology and sporulation in the disruption mutants, overview, the phenotype is reversed by expression of Sacharomyces cerevisiae chitin deacetylase CDA1