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EXPRESSION
ORGANISM
UNIPROT
LITERATURE
treatment of knock-in Gne p.M712T mouse model with N-acetylmannosamine, the Gne transcript expression, in particular mGne2, increases significantly, likely resulting in increased Gne enzymatic activities, especially in kidney and skeletal muscle
treatment of knock-in Gne p.M712T mouse model with N-acetylmannosamine, the Gne transcript expression, in particular mGne2, increases significantly, likely resulting in increased Gne enzymatic activities, especiallyin kidney and skeletal muscle
treatment of knock-in Gne p.M712T mouse model with N-acetylmannosamine, the Gne transcript expression, in particular mGne2, increases significantly, likely resulting in increased Gne enzymatic activities, especially in kidney and skeletal muscle
treatment of knock-in Gne p.M712T mouse model with N-acetylmannosamine, the Gne transcript expression, in particular mGne2, increases significantly, likely resulting in increased Gne enzymatic activities, especially in kidney and skeletal muscle
treatment of knock-in Gne p.M712T mouse model with N-acetylmannosamine, the Gne transcript expression, in particular mGne2, increases significantly, likely resulting in increased Gne enzymatic activities, especiallyin kidney and skeletal muscle
treatment of knock-in Gne p.M712T mouse model with N-acetylmannosamine, the Gne transcript expression, in particular mGne2, increases significantly, likely resulting in increased Gne enzymatic activities, especiallyin kidney and skeletal muscle