3.2.1.169: protein O-GlcNAcase
This is an abbreviated version!
For detailed information about protein O-GlcNAcase, go to the full flat file.
Word Map on EC 3.2.1.169
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3.2.1.169
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o-glcnacylation
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streptozotocin
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o-glycosylation
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nucleocytoplasmic
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beta-cell
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diabetogenic
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perfringens
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thiamet
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pugnacity
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tetratricopeptide
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thetaiotaomicron
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substrate-assisted
-
hexosaminidases
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medicine
- 3.2.1.169
-
o-glcnacylation
- streptozotocin
-
o-glycosylation
-
nucleocytoplasmic
-
beta-cell
-
diabetogenic
- perfringens
-
thiamet
-
pugnacity
-
tetratricopeptide
- thetaiotaomicron
-
substrate-assisted
- hexosaminidases
- medicine
Reaction
Synonyms
beta-N-acetylglucosaminidase, BtGH84, CP40, CpNagJ, CpOGA, cytoplasmic O-GlcNAcase, endo-beta-N-acetylglucosaminidase, EndoS-like endoglycosidase, glycoside hydrolase O-GlcNAcase, hexosaminidase, hOGA, mNCOAT, NagJ, NCOAT, neutral hexosaminidase C, neutral O-GlcNAcase, O-GlcNAc hydrolase, O-GlcNAc selective N-acetyl-beta-D-glucosaminidase, O-GlcNAc specific beta-N-acetylglucosaminidase, O-GlcNAc-selective N-acetyl-beta-D-glucosaminidase, O-GlcNAc-selective-N-acetyl-beta-D-glucosaminidase, O-GlcNAcase, O-linked beta-N-acetyl glucosaminase, O-linked N-acetylglucosaminase, OGA, OGA-FL, OGA-NV
ECTree
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General Information
General Information on EC 3.2.1.169 - protein O-GlcNAcase
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malfunction
enzyme knockout mice show nearly complete perinatal lethality associated with low circulating glucose and low liver glycogen stores. Enzyme loss leads to defects in metabolic homeostasis culminating in obesity and insulin resistance
malfunction
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enzyme loss in Drosophila globally impacts the epigenetic machinery allowing N-acetyl-D-glucosamine accumulation on RNA polymerase II and numerous chromatin factors including TRX, ASH1, and SET1