1.6.5.10: NADPH dehydrogenase (quinone)
This is an abbreviated version!
For detailed information about NADPH dehydrogenase (quinone), go to the full flat file.
Word Map on EC 1.6.5.10
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1.6.5.10
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neutrophil
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endothelial
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p47phox
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phagocyte
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apocynin
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dismutase
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artery
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gp91phox
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cardiovascular
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nicotinamide
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hypertension
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angiotensin
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catalase
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oxidases
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cardiac
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leukocyte
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phorbol
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monocyte
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mapks
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sod
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aortic
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granulomatous
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diphenyleneiodonium
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diphenylene
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pma
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iodonium
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rac1
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atherosclerosis
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chemiluminescence
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phagocytosis
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xanthine
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polymorphonuclear
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myristate
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microglial
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myeloperoxidase
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ii-induced
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dihydroethidium
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endothelium-dependent
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flavocytochrome
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zymosan
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peroxynitrite
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phagosomes
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tempol
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fmlp
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ros-dependent
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microbicidal
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nadph-oxidase
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nitrotyrosine
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lucigenin
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medicine
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fmlp-induced
- 1.6.5.10
- neutrophil
- endothelial
- p47phox
- phagocyte
- apocynin
- dismutase
- artery
- gp91phox
- cardiovascular
- nicotinamide
- hypertension
- angiotensin
- catalase
- oxidases
- cardiac
- leukocyte
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phorbol
- monocyte
- mapks
- sod
- aortic
- granulomatous
- diphenyleneiodonium
-
diphenylene
- pma
- iodonium
- rac1
- atherosclerosis
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chemiluminescence
-
phagocytosis
- xanthine
-
polymorphonuclear
- myristate
- microglial
- myeloperoxidase
-
ii-induced
- dihydroethidium
-
endothelium-dependent
-
flavocytochrome
- zymosan
- peroxynitrite
-
phagosomes
-
tempol
- fmlp
-
ros-dependent
-
microbicidal
- nadph-oxidase
-
nitrotyrosine
- lucigenin
- medicine
-
fmlp-induced
Reaction
Synonyms
AKR1C9, ArsH, azoreductase, CBR4, dehydrogenase, reduced nicotinamide adenine dinucleotide phosphate (quinone), EC 1.6.99.6, MdaB, More, NADPH oxidase, NADPH quinone oxidoreductase, NADPH quinone oxidoreductase 1, NADPH quinone reductase, NADPH-dependent quinone reductase, NADPH-quinone oxidoreductase, NADPH:quinone oxidoreductase 1, NQO1, PA1225, PpAzoR, XAC2229, Zta1p
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medicine
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arsenic exposure is associated with an increased risk of urothelial carcinoma, inorganic arsenic, monomethylarsonic acid, and dimethylarsinic acid, are determined in urothelial carcinoma patients from arsenic-contaminated areas of southwestern Taiwan, no correlation of the arsenic methylation capability of the patients and C609T genotype of NQO1 is found
medicine
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Pro187Ser polymorphism in NQO1 has a limited role in the development of Tardive dyskinesia (a potentially irreversible side effect of antipsychotic medication treatment that occurs in approximately 25% of chronically treated schizophrenia patients)